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Results 1-10 of 14 (Search time: 0.003 seconds).
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Issue DateTitleAuthor(s)
2018National survey: Evaluation of cardiovascular risk factors in Thai patients with type 2 diabetes and chronic kidney disease after the development of cardiovascular diseaseChangsirikulchai S.; Sangthawan P.; Janma J.; Sripaiboonkij N.; Rattanamongkolgul S.; Thinkhamrop B.
2018Effect of Magnesium on Glomerular Filtration Rate and Recovery of Hypertension in Women with Severe PreeclampsiaKreepala C.; Luangphiphat W.; Villarroel A.; Kitporntheranunt M.; Wattanavaekin K.; Piyajarawong T.
2018P2Y receptor regulation of K2p channels that facilitate K + secretion by human mammary epithelial cellsSrisomboon Y.; Zaidman N.A.; Maniak P.J.; Deachapunya C.; O’Grady S.M.
2018Vitamin D-binding protein and its polymorphisms as a predictor for metabolic syndromeKaruwanarint P.; Phonrat B.; Tungtrongchitr A.; Suriyaprom K.; Chuengsamarn S.; Schweigert F.J.; Tungtrongchitr R.
2018Suppression of Nrf2 confers chemosensitizing effect through enhanced oxidant-mediated mitochondrial dysfunctionSompakdee V.; Prawan A.; Senggunprai L.; Kukongviriyapan U.; Samathiwat P.; Wandee J.; Kukongviriyapan V.
2018Prevalence of vitamin D deficiency in cord blood of newborns and the association with maternal vitamin D statusAriyawatkul K.; Lersbuasin P.
2018Aspirin suppresses components of lymphangiogenesis and lymphatic vessel remodeling by inhibiting the NF-κB/VCAM-1 pathway in human lymphatic endothelial cellsPrangsaengtong O.; Jantaree P.; Lirdprapamongkol K.; Ngiwsara L.; Svasti J.; Koizumi K.
2018Shikonin suppresses lymphangiogenesis via NF-κB/HIF-1α axis inhibitionPrangsaengtong O.; Jantaree P.; Lirdprapamongkol K.; Svasti J.; Koizumi K.
2018P38 inhibitor inhibits the apoptosis of cowanin-treated human colorectal adenocarcinoma cellsChowchaikong N.; Nilwarangkoon S.; Laphookhieo S.; Tanunyutthawongse C.; Watanapokasin R.
2018Appropriateness of using tests for blood glucose and diabetic complications in clinical practice: Experiences in a hospital in ThailandLekskulchai V.