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Title: | Tuberculous peritonitis in a cerebral palsy patient: A challenge in diagnosis and management |
Authors: | Tangsirapat V. Chakrapan Na Ayudhya V. Kongon P. Chakrapan Na Ayudhya K. Sookpotarom P. Vejchapipat P. |
Keywords: | ceftriaxone metronidazole terbium tuberculostatic agent abdominal distension abdominal pain abdominal radiography abdominal tenderness Acinetobacter infection adult appendix perforation Article bacterium culture case report cerebral palsy clinical article computer assisted tomography dehydration diagnostic error fatigue fever hospital acquired pneumonia human large intestine disease leukocyte count linear energy transfer male mental development mental health Mycobacterium tuberculosis neutrophil count polymerase chain reaction priority journal spasticity tuberculous peritonitis vomiting young adult |
Issue Date: | 2019 |
Abstract: | Introduction: Diagnosis of tuberculous peritonitis (TBP) in a normal person, although possible, is often difficult to make because of its non-specific symptoms and signs. However, establishing a diagnosis of TBP in a patient with cerebral palsy (CP) does not seem to be possible due to impaired mental development accompanied by communication problems. Presentation of case: A 19-year-old spastic man diagnosed with CP presented with fever and a nonverbal complaint of abdominal pain. The conditions were hard to evaluate due to his mental status. Abdominal radiography showed dilatation of both small and large bowels, and a subsequent computed tomography (CT) scan did not provide any additional information. With respect to a common suspected cause, a diagnosis of perforated appendicitis was established. However, at the theatre, there was only bowel dilatation with multiple small nodules at the serosa of small and large bowels. Postoperatively, polymerase chain reaction and culture revealed Mycobacterium tuberculosis, thereby leading to a diagnosis of TBP. Discussion: Due to spasticity caused by CP, on examination, the patient presented with board-like rigidity, from which a diagnosis of a surgical condition was established. The misdiagnosis of an acute abdomen situation had let the patient to undergo an unnecessary exploration. To our knowledge, there has not been a report of TBP in a CP patient. Conclusion: The diagnosis of TBP had been complicated by the presence of CP in the reported case. The underlying CP not only preclude the diagnosis of TBP, but also produced symptoms that mimicked a condition requiring surgery. © 2019 The Authors |
URI: | https://ir.swu.ac.th/jspui/handle/123456789/12620 https://www.scopus.com/inward/record.uri?eid=2-s2.0-85064569340&doi=10.1016%2fj.ijscr.2019.04.019&partnerID=40&md5=ab9837385a552598fb04d2f90e6ed1bd |
ISSN: | 22102612 |
Appears in Collections: | Scopus 1983-2021 |
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